Risks with Periodontitis and Cardiovascular Disease
- Estimated Time: 120 minutes
- Credits: 2.0 CEU
- Passing Score: 80%
Course Overview
The link between oral health and heart health has moved from a hypothesis to a substantial body of scientific evidence. Periodontitis is no longer viewed as a localized infection of the gums but as a chronic inflammatory burden with far-reaching systemic consequences. This course examines the intricate relationship between periodontal disease and cardiovascular disease (CVD), exploring the biological mechanisms that connect the mouth to the heart. It evaluates the strength of the epidemiological data and distinguishes between correlation and causation in the context of atherosclerotic vascular disease.
The curriculum delves into the two primary pathways of pathogenesis: the direct invasion of arterial walls by oral bacteria via bacteremia, and the indirect systemic effects of chronic inflammation. Participants will learn how periodontal pathogens like Porphyromonas gingivalis have been isolated in atheromatous plaques and how the host's inflammatory response measured by biomarkers such as C-reactive protein (CRP) accelerates the formation of clots and arterial narrowing. The course also addresses shared risk factors, such as smoking and diabetes, that amplify the severity of both conditions simultaneously.
Finally, this course provides actionable strategies for the dental team to manage patients with comorbid cardiovascular risks. It reviews the potential benefits of non-surgical periodontal therapy in improving endothelial function and lowering systemic inflammatory markers. By adopting an interprofessional approach, dental professionals can play a pivotal role in the primary and secondary prevention of cardiovascular events, reinforcing the message that a healthy mouth is essential for a healthy heart.
Learning Objectives
After completing this course, participants will be able to:
- Summarize the epidemiological evidence linking moderate-to-severe periodontitis with an increased risk of atherosclerotic cardiovascular disease.
- Explain the “direct pathway” of pathogenesis, specifically how oral bacteria enter the bloodstream and colonize arterial walls.
- Describe the “indirect pathway” mediated by systemic inflammatory mediators like C-reactive protein (CRP), IL-6, and TNF-alpha.
- Identify the specific virulence factors of Porphyromonas gingivalis that promote platelet aggregation and atheroma formation.
- Analyze the impact of shared risk factors, such as tobacco use, obesity, and diabetes, on the progression of both diseases.
- Evaluate the current evidence regarding the effect of scaling and root planing on surrogate cardiovascular markers (e.g., endothelial function).
- Recognize the specific associations between periodontal disease and different forms of CVD, including myocardial infarction, stroke, and hypertension.
- Implement a patient education protocol that effectively communicates the oral-systemic risk without causing unnecessary alarm.
This course is members-only
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